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Interference of Severe Idiopathic Scoliosis in the Respiratory and the Stomatognathic System in Women

Journal Name:

  • International Journal of Science and Engineering Investigations

Publication Year:

  • 2017

Key Words:

Author NameUniversity of Author
Abstract (2. Language): 
The present study aimed to analyze the changes in the respiratory and the stomatognathic system caused by acute idiopathic scoliosis. Methods: Twenty women, aged 18 to 30 years (23.80 ± 2.94 years) were divided into two groups: Scoliosis Group (n=10) with acute idiopathic scoliosis and Control Group (n=10) with healthy participants. The masticatory muscle activity and the muscle activity of the spine were evaluated by means of electromyography. A manuvacuometer was used to measure the respiratory muscle strength to understand how the disorders caused by severe idiopathic scoliosis in the spinal system can affect the performance of both the stomatognathic and the respiratory system. Electromyographic and respiratory muscle strength data were tabulated and submitted to statistical analysis (SPSS 21.0) using the Student’s t test for independent samples, with a significance level of 5% and a 95% confidence interval. Results: The results obtained showed that the convex side of the curve in severe idiopathic scoliosis has produced functional changes in the stomatognathic system, increasing the action potential of the masseter, temporal and sternocleidomastoid muscles. The results showed that the strength generated by the inspiratory and expiratory muscles was reduced by the influence of the action potential of the multifidus muscles that create rotational motion between vertebras. Conclusion: It can be concluded that acute idiopathic scoliosis has interfered in the clinical conditions of mandibular rest, chewing raisins and peanuts, neutral cervical posture, spine extension and respiratory muscle strength
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International Journal of Science and Engineering Investigations, Volume 6, Issue 60, January 2017 72
www.IJSEI.com Paper ID: 66017-08
ISSN: 2251-8843
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