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YAYGİN ORAL LEUKOPLAKININ TEDAVİSİNDE ND-YAG LASER (BİR OLGU RAPORU)

TREATMENT OF WIDESPREAD ORAL LEUKOPLAKIA WITH ND-YAG LASER (A CASE REPORT)

Journal Name:

  • İstanbul Üniversitesi Diş Hekimliği Fakültesi Dergisi

Publication Year:

  • 1997

Key Words:

Keywords (Original Language):

Author NameUniversity of AuthorFaculty of Author
Abstract (2. Language): 
Oral leukoplakia is the most common (% 85) premalig-nant lesion which İs seen in oral mucosa and İt has been reported that malignant transformation rate is 4-5 percent. Although lesion may be found anywhere in the oral cavity, the buccal mucosa and commissures were most frequently involved, followed in descending order by alveolar mucosa, tongue, lip, hard and soft palates, floor of the mouth and gingiva. The lesions of oral leukoplakia show considerable variation in size, location and clinical appearance. This lesion is more common in men man in women and that it is seen chiefly over the age of 40. The etiology of leukoplakia to be a varied one. Tlie initiation of the condition may depend not only upon extiinsic local factors, but also upon intrinsic predisposing factors. Factors most frequently blamed have been tobacco, alcohol, oralsepsis, local irritation, syphilis, vitamin deficiency, endocrine disturbances and galvanism. Elimination of local factors have been sufficient in the treatment of oral leukoplakia but conservative surgical excision, electro-coagidation and laser surgery have been used in persisted lesions. In our presented case, a 46 year old men referred to our clinic with the complaint of persistent and widespread lesions in his maxillary and mandibulary crest. After the clinical and histopathological examination the lesions were successfully treated with the laser surgery.
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Abstract (Original Language): 
Oral leukoplaki, oral mukozada görülen en yaygın (% 85) prekanseröz lezyondur ve malign transformasyonun % 4-5 olduğu belirtilmektedir, Lezyon oral kavite-nin her yerinde ve sıklıkla bukkal mukoza ve kommis-surlarda lokalize olmaktadır. Bunu görülme sıklığı sırasıyla; alveoler mukoza, dil, dudak, sert ve yumuşak damak, ağız tabanı ve gingiva izlemektedir. Oral leukoplaki lezyonları oldukça farklı boyut, lokalİzasyon ve klinik görüntü vermektedir. Lezyon erkeklerde kadınlardan daha sık ve özellikle 40 yaş üzerinde görülmektedir. Leukoplakinin etyolojisi çok değişkendir. Lezyonların oluşması sadece (dışarıdan gelen) lokal faktörlere bağlı olmayıp aynı zamanda (İçeriden gelen) predispozan faktörlerede bağlı olabilir. Tütün, alkol kullanımı, oral sepsis, lokal irritasyon, vitamin eksikliği, hormonal bozukluklar ve galvanik akım gibi faktörler sıklıkla sorumlu olmaktadır. Lokal faktörlerin elimine edilmesi yeterli olmakta ancak ısrarlı lez-yonlarda konservatif cerrahi eksizyon, elektrokoter ve laser cerrahisi uygulanmak tadır. Sunulan olguda, 46 yaşında erkek hasta maksiller ve mandİbuler kretlcr üzerinde ısrarlı ve yaygın lezyonlardan yakınarak kliniğimize başvurdu. Klinik ve histopatolojik inceleme sonrası lezyonlar başarılı bir şekilde laser cerrahisi ile tedavi edildi.
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  • Turkish

REFERENCES

References: 

1. Abbey LM. Precancerous lesions of the mouth. Curt OpinDent 1991; 2: 773-776.
2. Axel! T. The professional role* of the dentist under the aspects of precancer and cancer diagnosis and management. Lit Dental J1993; 45:609-611.
3. Bouquot JE. Reviewing oral leukoplakia: clinical concepts for the 1990S. JHDA 1991: 222:80-82.
4. Cliiesa F ve ark. Follow - up of oral leukoplakia after carbon dioxide laser surgery, Arc Otolaryngol Head Neck Surg 1990: 226:117-180.
5. Cooke BEC. Leukoplakia buccalis and epithelial nae-vi. A clinical and histological study. Br J Dermatol 1956.
6. Darling MR, Arendorf TM. Effects of cannabis smoking on oral soft tissues. Community Dent Oral Epidemiol 1993:22:78-81.
7. FramajW. The management of oral premalignant lesions. British Journal ofOral & Maxillofac Surg 1992:30:71.
8. Gasper L. The use of the high power lasers in oral surgery. Acta Biomed Ateneo Patínense 1992: 65:85-89.
9. Gendelman H., Actis A.B., Ouri H.O. Nc-odmium-YAG and C02 lasers in the treatment of pre-cancereous
lesions of the oral cavity. Acta Stomatol Belg 1993: 90:95¬101.
10. Grant WE, Hooper C, Speight PM, Macrobert AJ, Bown SG. Photodynamic therapy of malignant and premalignant lesions in patients with field cancerization of oral cavity. JLaryngol Otol 1993: 207:1140-1145.
11. Renstrup G. Leukoplakia of the oral cavity. Ada Odontoi Scand 1958: 26:99-100.
12. Roodenburg JLN, Panders AK, Vermey A. Carbon dixoide laser surgery of oral leukoplakia. Oral Stag Oral Med Oral Pathol1991: 72:670-674.
13. Shafcr WG, Mine MK, Levy BM. A textbook of oral pathology. 4th. cd. Philadelphia: WB Saunders. 1983: 86¬229.
14. Siclair Rj, Sinclair PJ. Carbon Dioxide laser in the treatment of cutaneous disorders. AustralasJ Dermatol 1991: 32:165-171.
15. Waldron CA, Shafcr WG. Current concepts ol'lcu-koplakia. hit DentJVJGO: 20:350-360.
16. WilliamsJL. Oral cancer and precancer: clinical features. Br Dent J1990: 26»: 13-17.

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