You are here

Home » Content

Herbisidlere maruz kalma ile koroner arter ektazisi arasındaki ilişki

The relationship between exposure to the herbicides and coronary artery ectasia

Journal Name:

  • Süleyman Demirel Üniversitesi Tıp Fakültesi Dergisi

Publication Year:

  • 2007

Key Words:

Keywords (Original Language):

Author NameUniversity of Author
Abstract (2. Language): 
The dilatation of the epicardial coronary arteries 1.5-2 times that of normal adjacent segment is called coronary ectasia and >2 times that of normal adjacent segment is called coronary aneurysm. Longterm exposure to the herbicides, which are frequently used by farmers, cause relaxation in the vascular smooth muscles via nitric oxide by increasing acetilcholin concentrations. However, it is unknown whether chronic exposure to these substances causes coronary ectasia. Therefore, the aim of the present study was to compare the rate of exposure to these herbicides in patients with coronary ectasia and control group.
Bookmark/Search this post with
Abstract (Original Language): 
Anjiyografik olarak, epikardiyal koroner arterlerde obstrüktif lezyon olmaksızın normal luminal çaptan 1.5¬2 kat arasında genişleme koroner ektazi, 2 kattan fazla genişleme ise koroner anevrizma olarak tanımlanmaktadır. Çiftçilerin yaygın olarak kullandıkları herbisidlere uzun süre maruz kalınması asetilkolin konsantrasyonunu artırarak nitrik oksit üzerinden vasküler düz kaslarda relaksasyona neden olmaktadır. pakat kronik relaksasyonun koroner arter ektazisine neden olup olmadığı bilinmemektedir. Çalışmamızın amacı, izole koroner arter ektazisi olan olgularda kontrol grubuna göre herbiside maruz kalma sıklığını araştırmaktı.
FULL TEXT (PDF): 
PDF icon arastirmax-herbisidlere-maruz-kalma-ile-koroner-arter-ektazisi-arasindaki-iliski.pdf
  • 3
13-16
  • Turkish

REFERENCES

References: 

1. Hartnell GG, Parnell BM, Pridie RB. Coronary artery ectasia, its prevalence and clinical significance in 4993 patients. Br Heart J 1985; 54: 392-95.
2. Krüger D, Stierle U, Herrmann G, Simon R, Sheikhzadeh A. Exercise-Induced myocardial ischemia in isolated coronary artery ectasias and aneurysms ("Dilate coronaropathy"). Am J Cardiol 1999; 34: 461-70.
3. Sayın T, Döven O, Berkalp B, Akyürek Ö, Güleç S, Oral D. Exercise-induced myocardial ischemia in patients with coronary artery ectasia without obstructive coronary artery disease. Int J of Cardiol 2001;78: 143-49.
4. MarkisJE, JoffeCD, CohnPF,FeenDJ,Herman MV, Gorlin R. Clinical significance of coronary artery ectasia. Am J Cardiol 1976; 37: 217-22.
5. Maehara A, Mintz GS, Ahmed JM, Fuchs S, Castagna MT, Pichard AD, Datler LF, Waksman R, Suddath WO, Kent KM, Weissman NJ. An ıntravascular ultrasound classification of angiographic coronary artery aneurysms. Am J Cardiol2001;88:365-70.
6. Sorrel VL, Davis MJ, Bove AA. Origins of coronary artery ectasia. Lancet 1996; 347: 136¬37.
7. Kahraman H, Ozaydin M, Varol E, Aslan SM, Dogan A, Altinbas A, Demir M, Gedikli O, Acar G, Ergene O. The diameters of the aorta and its major branches in patients with isolated coronary artery ectasia. Tex Heart Inst J. 2006;33(4):463-
8.
8. Williams MJA, Stewart RAH. Coronary artery
S.D.Ü. Tıp pak.
Derg
. 2007:14(3)/ 13-16
16
Özaydın, Koroner ektazide herbisidler
ectasia, local pathology or diffuse disease. Cathet Cardiovasc Diagn 1994; 33: 116-19. 9. Kajinami K, Kasashimas S, Oda Y, Koizumi J, Katsuda S, Mabuchi H. Coronary ectasia in familial hypercholesterolemia:histopathologic study regarding matrix metalloproteinases. Mod Pathol 1999; 12: 1174-80.

Thank you for copying data from http://www.arastirmax.com