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KÖK ÇÜRÜKLERİ VE TEDAVİLERİ

ROOT CARIES AND TREA TING ROOT SURFACE CARIES

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Abstract (2. Language): 
Root caries is a soft rrreguaarly shaped lesion either totally confined to the root surface or involving the undermining of enamel at the cemento enamel junction, but ciinical/y indicating that the lesion initiated on the root surface. Root caries can be initiated only if the root surface is exposed to the oral envrroment. The risk of root caries increases with inadequate oral hygiene and nn reductions in salivary flow rate. Many root caries lesions do not need restorative treatment. Accessible, shallow lesions can be made caries free and easy to clean through caries depridement with hand nnstruments, finishing burs and\orpolishing disks. Arrested lessions with a hard to leathery surface are often amenable to treatment with topikai flourides in combination with a chlorhexidine rince. When root caries has progresed such that restoration of loast structure is necessary they can restored with different restorative materials. Typical root carious lesions are difficult to restore because the gingiva/ margin of the lesion is often subgingival. To gain acces to this area a variety of techniques have been developed. In this article etiology and incidence of root surface caries described and summarizes the effectiveness of restorative materials used to restore root surfaces.
Abstract (Original Language): 
Kök çürüğü klinik oaarak kök yüzeyinden başlayan kök yüzeyinde sınırlı veya mine sement bileşimindeki minenin attında kaplayan yumuşak düzensiz şekilli lezyondur. Kök çürüğü sadece kök yüzeyinin ağız ortamına açıldığı durumlarda baş/ar. Yetersiz ağız hijyeni ve tükürük akış hızında azalma olduğunda kök çürüğü riski oluşumu artmaktadır. Kök çürük lezyonlarının bir çoğu restoratif tedavi gerektirmemektedir. U/aşılabilir sığ lezyon/ar el aletler, bitirme frez/eri ve\veya paraatma diskleri yardımı ile çürükten çürüksüz ve kolay temizlenebilen alanlara dönüştürülebilir. Sert kayışımsı yüzeyler şekinnde olan durağan lezyonaar k/orheksidin ile birlikte topikal florid uyguaamaaarı ile birlikte tedavi edilebilir. Kök çürüğü doku kaybıyla birlikte ilerlediğinde farklı restoratif materyaller l/e restorasyonu gerekmektedir. Kök çürüklerini restore etmek, lezyonun dişeti sınırı çoğunlukla subgingival alanda son/andığından güçtür. Bu aaana ulaşabilmek için değişik teknikler geiiştiri/miştir. Bu derlemede kök çürüklerinin etyolojisi ve görülme sıklığından bahsedilmiş ve kök çürüklerinin restorasyonunda kullanılan restoratif materyallerin etkinliği özetlenmiştir.
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REFERENCES

References: 

1. Al-Khateeb S, Angma-Mansson B, DeJosseinn E. In vivo quantification of changes in caries lesions in orthodontic patients. J Dent Res 1996; 75:127¬131.
2. Ando M, Analoui M, Schemehorn BR. Comparison of lesion analysis by microradiograpy and con focal microscopy. J Dent Res 1995; 74:48-52.
3. Atkinson JC, Wu AJJ. Salivary gland dysfunction: causes, symptoms, treatment. J Am Dent Assoc 1994; 125:409-416.
4. Banting DW, Elen RP, Filery ED. A longitudinal study of root caries: baseline and insidence data. JDent Res 1985; 64:1141-1144.
5. Baysan A, Prnnz JF, Lynch E. Clinical criteria used to detect primary root caries with e/ectrical and mechanical measurements nn vitro. Am J Dent 2004; 17:94-98.
6. Baysan A, Lynch E, El/wood R, Davies R, Petersson L, Borsboom P. Reversal of primary root caries using dentrifices containing 5000 and 1100 ppm flouride. Caries Res 2001; 35:41-6.
7. Baysan A, Lynch E. Effect of ozone on the oral microbata and clinical severity of primary root caries. Am J Dent2004; 17:56-60.
8. Beck JD. The epidemiology of root caries: North American studies. Adv Dent Res 1993; 7:42-51.
9. Begghton D, He/Iyer PH, Lynch EJ, Heath MR. Salivary levels of mutans streptococci, lactobacilli, yeast and root caries prevelance in non-institutionalized eldery dental patients. Communtty Dent Oral Epidemiol 1991; 219:302¬307.
10. Begghton D, Lynch E, Heath MR. A microbiological study of primary root caries lesions with different treatment needs. J Dent Res 1993; 72:623-629.
11. Burgess JO, Gal/o JL. Treating root surface caries. J Dent Cinn N Am 2002; 46:385- 404.
12. Chan DCN. Modified matrix adaptation for subgingival class II amalgam restorations. Oper Dent 2003; 28: 469-472.
36
Atatürk Üniv. Diş Hek. Fak. Derg. Cilt:17, Sayı: 1, Yıl: 2007, Sayfa: 32-38
13. De Joseinn De Jong E, Sundstrom F, Angmar-Mansson B, Ten Bosch JJ. QLF-vision: reproducibilttyof in vivo quantification of enamel mineral loss. J Dent Res 1994; 73:200-205.
14. De Joseinn De Jong E, Sundstrom F, Westering H. A new method for in vivo quantffcation of changes in initial enamel caries with laser fluorescence. Caries Res 1995; 29:2-7.
15. Dodds M, Suddick R. Caries risk assessment for determination of focus and nntenstty of prevention nn a dental school clinic. J Dent Educ 1995; 59:945-956.
16. Em/son CG, Lindquist B, Wennerholm K. Recolonzzation of human tooth surfaces by streptococcus mutans after suppression by chlorhexidine treatment. J Dent Res 1987; 66:1503-8.
17. Em/son CG, Rava/d N, Brrkhed D. Effects of a 12-month prophylactic programme on selected oral bacterial populations on root surfaces with active and inactive carious lesions. Caries Res 1993; 27:195-200.
18. Em/son CG. Potential efficacy of chlorhexidine against mutans streptococci and human dental caries. J Dent Res 1994; 73:682-91.
19. Featherstone JDB. Flouride, remnneraizzation and root caries. Am J Dent 1994; 7:271-4
20. Fejerskov O, Luan WM, Nyvad B Active and inactive root surface caries lesions in a selected group of 60-90 year old Danes. Caries Res 1991; 25:385-391.
21. Ferreira AG, Analoui M, Ando M. Using dye enchanced QLF for anayznng incipient lesions. J Dent Res 1995; 74:192.
22. Fure S, Romaniec M, Em/son CG, Krasse B. Proportions of Streptococcucs mutans, Lactobasilli and Actinomyces spp in root surface plaque. Scan J Dent Res 1987; 95:119-123.
23. G/iboa I, Cardash H. A conservative technique for restoring a tooth affected by interproximal root cares. J Prosthet Dent2003; 89:221-2.
24. Gwinett J. Moist versus dry dentin: its effect on shear bond strength. Am J Dent 1992; 5:127.
25. Imazato S, Ikebe K, Nokubi T, Ebisu S, Walls AWG. Prevalence of root caries in a selected population of older adults in Japan. J Oral Rehab 2006; 33:137-143
ELİGÜZELOĞLU, ÖZCAN
ÜÇTAŞLI, ÖMÜRLÜ
26. James B. Summttt, J. Wiliaam Robbnns, Richards S. Schwartz. Fundamentals of Operative Dentistry Second Edition, A Contemporary Aproach. Quintessence Pubishnng Co, Inc 2001:365-377
27. Jones JA. Root caries: prevention and chemotherapy. Am J Dent 1995; 8:352-357.
28. Jordan HV, Hammond BF. Filamentous bacteria isolated from human root caries. Arch Oral Biol 1972; 17:1333-1342.
29. Katz RV, Hazen SP, Chitton NW, Muma RD Jr. Prevalence and nntraoral distrubution of root caries in an adutt population. Caries Res 1982; 16:265-271.
30. Katz S. The use of flouride and chlorhexidine for the prevention of radiation caries. J Am Dent Assoc 1982; 104:164-70.
31. Katz RV. The clinical diagnosis of root caries; issues for the clinicans and researcher. Am J Dent 1995; 8:335-341.
32. Kettjens HM, Schaeken M J, Van Der Hoevens JS, Hendrllos JC. Microflora of plaque from sound and carious root surfaces. Caries Res 1987; 21:193¬199.
33. Locker D, Slade GD, Leake JL. Prevelance of and factors associated with root decay in older adutts nn Canada. J Dent Res 1989; 68:768-772.
34. Loe H, Rnndom- Schiott C, Glavind L, Karrnng T. Two years oral use of chlorhexidine in man. J Periodont Res 1976; 11:135-44.
35. Luan WM, Boelum V, Chen X. Dental caries nn adutt and elderly Chinese. J Dent Res 1989;68:1771-1776.
36. Lynch E. Relationship between cinnical crtteraa and microflora of primary root caries. Early Detection of Dental Caries. Cincinnati, OH; Sidney Printing Works. 1996:195-242.
37. Lynch E, Baysan A. Reversal of primary root caries using a dentifrice with a high flouride content. Caries Res 2001;35:60-64.
38. Mackenzie IC, Nuki K, Loe H, Rnndom Schiott C. Two years oral use of chlorhexidine in man .V: Stratum corneum of oral mukosa. J Periodont Res 1976; 11:165-71.
39. Mjor JA. Secondary caries: a literature review with case reports. Quint Int 2000; 31:165-79.
40. Nakabayashi N, Nakamura M, Yasuda N. Hibrtt layer as a dentin bonding mechanism. J Esthet Dent 1991; 3:133-8.
37
Atatürk Üniv. Diş Hek. Fak. Derg. Cilt:17, Sayı: 1, Yıl: 2007, Sayfa: 32-38
ELİGÜZELOĞLU, ÖZCAN
ÜÇTAŞLI, ÖMÜRLÜ
41. Newttter DA, Katz RV, Cive JM. Detection of root caries: sensitivity and specivtty of a modified explorer. Gerodontics 1985; 1:65-67.
42. Roberson TM, Lundeen TF. Cariology: The lesion, etiology, prevention and control. Sturdevants's Art & Science of Operative Dentistry Fourth Edition Mosby, St. Louis, 2002:110
43. Schupbach P, Osterwalder V, Guggenheim B. Human root caries: microbiota of a iimited number of root caries lesions. Caries Res 1996; 30:52-64.
44. Sma/es RJ, Wetherell JD. Review of bonded amaggam restorations and assesment nn a general practise over 5 years. Oper Dent 2000; 25:374¬81.
45. Socransky SS, Hubersak C, Propas D. Introduction of periodontal destruction nn gnotobiotic rats by a human oral strain of Actinomyces naeslundii. Arch Oral Biol 1970;15:993-995.
46. Summtt JB, Burgess JO, Berry TG, Osborne JW, Robbnns JW, Haveman CW. The performance of bonded vs. pin-retained complex amalgam restorations:a five year clinical evaluation. JADA 2001; 132:923-31.
47. Summtt JB, Robbnns JW, Schwartz RS Operative Dentistry
48. Tay FR, Pashley DH. Resin bonding to cervical sclerotic dentin: A review. J Dent 2004; 32: 173¬196.
49. Van Strjp AAJ, Van Steenbergen TJ, Ten Cate JM. Bacterial colonization of mineralized and completely demnneraizzed dentine in situ. Caries Res 1997; 31:348-355.
50. Watanabe MGC. Root caries prevalence nn a group of Braziiaan adutt dental patients. Braz Dent 2003; 14:153-6

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