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RENAL TRANSPLANTASYON SONRASINDA OLGULARIN RENAL TUBULER ASÎDOZ YÖNÜNDEN İNCELENMESİ

THE EVALUATION OF RENAL TUBULER ACIDOSIS FALLOWING RENAL TRANSPLANTATION

Journal Name:

  • Türk Nefroloji, Diyaliz ve Transplantasyon Dergisi

Publication Year:

  • 1996

Key Words:

Keywords (Original Language):

Author Name
Abstract (2. Language): 
42 living related, 33 cadaveric; totaly 75 renal transplanted patients were studied in point of renal tubuler acidosis during their follow up period. 41 pa¬tients (% 54.66) who showed complet and incomplet renal tubuler acidosis classified as type-1, type-II and type-IV based on biochemical parameteres. The cases were groupped as the patients with acidosis, without acidosis and type-1, type-II, type-IV renal tubuler acidosis. Each of these group of patients serum creatinin levels, proteinuria, hypertension, rejection periods, serum Cyc. A levels, tissue compatibility, hyperparatyroidism, type of transplantation, trans¬plantation duration compared to each other istatis-ticly and no significant correlation were found bet¬ween them (p>0.05). As it is written before patogenesis of renal tubuler acidosis following renal transplantation is multifac-torial. It is very difficult to find out only one respon¬sible reason. It can appear any time almost every af¬ter transplantation and most of the time without trea¬ting it can easily dissappear.
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Abstract (Original Language): 
42'si canlı vericiden, 33ü kadavradan olmak üzere böbrek nakli yapılan toplam 75 olgu renal tubuler asidoz (RTA) yönünden incelendi. Komplet veya in-komplet asidoz saptanan 41 olgu (% 54.66) tip-I, tipli ve tip-IV RTA yönünden irdelendi. Asidozu olan ol¬gularla proteinüri, hipertansiyon, red sayısı, cyclos-porin-A (CycA) düzeyleri, doku uyumu, hiperparati-roidi, transplantasyon cinsi ve yaşı arasındaki ilişki araştırıldı ve istatistiki olarak anlamlı bir ilişki saptanmadı (p>0.05). Transplantasyon sonrası RTA'un tek bir patogenezden ziyade multifaktöriyel olabilece¬ği ve hemen her transplantasyon sonunda bir dönem ortaya çıktığı sonucuna varıldı.
FULL TEXT (PDF): 
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  • 1
26-30
  • Turkish

REFERENCES

References: 

1. Gyory AZ, Stewart JH, George CRP, et al. Renal tubuler acidosis, acidosis due to hyperkalemia, hypercalcemia, disordered citrate metabolism and other tubuler dysfuncti¬ons following human renal transplantation. Q J Med 1969; 38: 231-254.
2. Wilson DR, Siddiqui AA. Renal tubuler acidosis after kidney transplantation. Natural history and significance. Ann Intern Med 1973; 79: 352-361.
3. Kurtzman N. Acquired distal renal tubuler acidosis. Kid¬ney Int 1983; 24: 807-819.
4. Batlle DC, Moses MF, Manilgiod J et al. The pathogene-sis of hyperchloremic metabolic acidosis associated with
transplantation. Am J Med 1981; 70: 786.
5. Heering P, Grabensee B. Influence of ciclosporin A on renal tubuler function after kidney transplantation. Neph-
ron 1991; 59: 66-70.
6. Wrong O. Distal renal tubuler acidosis: The value of uri¬nary pH, pC(>2, and NH+4 measurement. Pediatr Nephrol 1991; (Germany) 5(2) p 249-55.
7. Rothstein M, Obialo C, Hruska KA. Renal tubuler acido-sis. End And Met. Clinics of North America 1990; Vol. 19, No: 4.
8. Eleanor L, Ramos Nicholar, L. et all. Clinical aspects of renal transplantation. Brenner MD, Rector Jr. MD. Kid¬ney W.B Saunders Company, Philadelphia, p: 2390
(1991).

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